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About the Endobiontic Nature of the Cattle-Leucosis

©Copyright 1997 by Enderlein Enterprises
by Professor Dr. Günther Enderlein, Germany
(Explore Issue: Volume 9, Number 3)

To an ever higher measure we find confirmation of the insight that the endobiontic portion of the bipolar complexes of chronic disease (Psora) shows its appearance through the totality of mammals. The amazing significance of this has been recognized one and a half centuries ago by Hahnemann, and he named it the "thousand headed hydra". The knowledge of the very earliest primal cultures that the conditional causes of diseases (the chronic complex of humoral pathology) is rooted in the life-style and especially in the diet, is confirmed throughout by the fact that domestic animals near humans are primarily subject to chronic diseases of the Endobiosis (congestive diseases), according to the most modern findings, and they are caused by the artificial fertilizers in the soil.

As for the microbiotic factors of true causes -- in contrast to the apparent health of civilized people, the total extent of mammals stands far superior in its original purity (with just the exception of domestic animals), according to the classic experiments with rats by the English doctor in India, MacCarrison.

Entirely in contrast to the Monomorphism of the Doctrine and of fundamental importance for our agriculture, stands out the absence of a "paroled" attitude toward the causal factors of micro-conditioned parasites and their far-reaching developmental cycle and the retreating of monomorphic fantasies, which would contradict the fermentation-researcher in his daily experiences (cf. Professor Dr. phil. Wilh. Henneberg, (Kiel); which also appears as natural fact to the biological soil researcher of today after the fundamental insights of a Professor Dr. F. Loehnis (Washington USA; Studies in the Life-cycle of Bacteria, in: Memoirs of the Nat. Academy of Sciences, Washington, Vol. 16, 1921); to which also the Veterinary-biologist would bring a far-reaching understanding, as is the case at the Institute for Animal Health, director Dr. med. vet. Tilgner in Kiel, with all his researchers, in exemplary ways. Because here the (by Professor Dr. med. Pierre Dolore of Lyon) demanded purely biological attitude for Health Research comes singularly into universal significance; its fundamental importance for total health in the akmic sense and the persistence of the human race have been recognized by the French Government by calling him into the ministerium and thus doing the matter justice.

Thus, Enderlein in 1916 had founded teaching of the cyclic developmental history of microbes and has found extensive grounding of this within four decades, inclusive of general biology. The fake existence of Monomorphism which is kept alive through "Paroles" is definitely threatened even in its last resort in Central Europe; for some districts, such as Hamburg and especially Schleswig-Holstein, there is already sufficient clarity. In the remainder of Europe one can choose from an abundance of pleomorphic researchers such as: Almquist, Freiherr von Klinckowstroem and Bartels (Sweden), A Cunningham (England), Colvee and Socias (Spain), K. von Neergaard and Spengler (Switzerland), J. Lominski (Poland). Regarding Russia and Asia: pleomorphism prevails there without any "Paroles", as Dr. med. Rabuchin (Moscow) has documented.

Through the Institute for Animal Health in Kiel, I have received a larger number of blood samples and blood smears of cattle, some having Cattle-Leucosis, some being healthy.

Already after the first few examinations, I was convinced that this to me fully unknown disease is of endobiontic nature. In the blood picture, it stands nearest to cancer and Hodgkin. Dr. Tilgner then actually confirmed for me after review of the microscopic preparations here in Aumuehle, that leucosis manifests even muscular and glandular swellings in cattle even if they do not reach the size of tumors as in human cancer.

The accumulation of Erythrocytes which are attacked by Symprotites in the Chondrite-stage up to the sporoid Symprotites with their net-like or grain-like appearance like onto dried albumin, increases here as in cancer until there are net-like infestation-forms, called "Reticulocytes" in the doctrine. But its endobiontic nature can be easily proven in every preparation because there are always marginal Symprotites (marginal grains, in the doctrine) found which project from the lumen of the Erythrocytes and then repeat flagellum-like or as Dendrites the typical form of the Chondrite-stage. This is shown in picture #4. While in cancer one cannot observe the take-up of Chondrites and especially the Symprotites from the Leucocytes and the Lymphocytes successfully by fully freeing the Leucocytes of these parasitary formations, in general, such clearings happen more frequently in cattle diseased with Leucosis. The further development of marginal Symprotites into bacterial rods in the catatact Ascite-phase also occurs; also the expelling of Symprotites living in the interior of the Erythrocyte which develop further into typical Thrombocytes during the expelling or sometimes even prior to it. They are special forms of Thecites of the cycle of parasites, that is, of the Endobiont (Mucor racemosus Fresen). The not rare high-valence Symprotite-grains of the size 1/4 to 1/3 of the Erythrocyte diameter, found in the more or less stricken Erythrocytes in cancer, have not been observed in leucosis of cattle up until now.

As for the changes in the white bloodcells in the course of intensification of their leucosis in cattle, forms appear in Leucocytes and also Lymphocytes according to dynamo-valance which destroy the nucleus and the cell-plasma, and through absorption of albumin they totally finish them without residue. In this process, the albumin constituents of the host cell are progressively replaced by the parasites in the Chondrite-stage until, finally, the original particles have changed more and more to the Chondrite-stage and the original living host cell has changed into a merely apparent existence. And this also when one believes that one can identify the apparent nuclear and cellular plasma.

In Figure 1, a Monocyte is pictured, the nuclear material of which still showing a nuclear position, which however has no longer any host material but consists, to the largest portion, of balled together parasitic materials of a Symplast, which contains all possible appearance forms of the Chondrite-stage. The easily visible, relatively small vacuola containing the lumen of the fake-nucleus offer a few remnants for understanding these balled complexes from the Dendroids of the Chondrites. The cell plasma of this Monocyte is already dispelled and changed into the material of the Chondrite-net, in which one can observe in fine detail the constant change of both comparative-morphologic units, namely of Symprotites (primal core) and of Fila, the flagellum-like threads.

 
Figure 1: Blood of Cattle-Leucosis. Monocyte with tiny vacuola. From the fake-nucleus, the tree-shaped Dendroid of the Chondrite-stage of the parasite (cattle #65a) are spreading out.   Figure 2. Blood from Cattle-Leucosis (cattle # 58a). A Leucocyte that is totally wasted by parasites (Endobionts). Nucleic material of Symplast in the Chondrite-stage with a few vacuola, strongly deformed. Plasma-materials totally transformed into the tree-shaped netting material of the Chondrite. The Symprotites mostly maintained, not disappeared due to thick growth of the filum. On the right, below, two Erythrocytes stricken by Chondrites.

Figure 2 shows a pictorial representation of a Leucocyte, the nuclear- and cell-plasma of which have been completely destroyed by the parasite. The fake-nucleus contains several larger vacuola and is, itself, far deformed. The material of the cell-plasma is transformed into a wide-looped net of Chondrites, the Symprotites of which have enlarged themselves partly; thus, they already have the tendency to develop further into Thrombocytes. On the right, below, two Erythrocytes are stricken by Chondrite-netting. On the left, above, a Chondrite-filum projects with end-head (Symprotite) freely beyond the Dendroit-complex; below in the center are a number of shorter ones.

A somewhat further diverted Leucocyte-remainder in the commencing break-up is shown in Figure 3. The remainder of the fake-nucleus is already falling apart into separate pieces, partly with small vacuola. Dendroid-fila are partly scattered in larger Symprotite-chains, which in the left center have already expelled themselves and have changed into Thrombocytes.

A further stage of decomposition of the Leucocyte changed into parasitic material is sown in Figure 4. Nuclear remains with larger and smaller vacuola; only insignificant remnants remain of the plasma-dendroid. In contrast, the remaining Symprotites of the parasite from the left cell-plasma-remainder have changed in larger numbers into thrombocytes.

 
Figure 3. Blood of Cattle-Leucosis (Cattle #65a). Leucocyte beginning to fall apart. Among the 17 surrounding Erythrocytes, 6 are not stricken by the Chondrite-net of the cell-plasma. Fake nucleus remainder torn apart and fissured by numerous small vacuola. Symprotites (primal cores) already in enlarged valence (in larger form) and partly already filling in the length of the fila in chain-formation. Partly already detached and in transmutation to Thrombocytes (left center); they are characterized by tiny granulation by having generally three to six or seven primal cores (Mych).   Figure 4. Blood from Cattle-Leucosis of 3/24/54 (Cattle #65a). Parasitic Symplast of the former nuclear material has larger and smaller vacuola. At the right, the still remaining remnant of the Chondrite-Dendroids; at left, the Symprotites have changed into Thrombocytes in greater numbers. Above the Leucocyte-remnant lies a Monocyte, which has become nearly freed of parasites through the Erythrocytes in their surroundings. Therefore, the Erythrocytes in the close surroundings are especially heavily stricken.

 

Above this Leucocyte-remnant is found a Monocyte; in its surroundings, the Erythrocytes have taken over the Chondrites of the parasites which had stricken it heavily, so that the Monocyte has become freed from these parasites through the Erythrocytes. This process can often be observed in Cattle-Leucosis, and very frequently in the case of the human 'Felty-Syndrome". This purificatory act occurs extremely rarely in cancer with such strength, so that the Erythrocytes are not successful in freeing the Leucocyte or Lymphocyte from parasites.

The Leucocyte shown in Figure 5 is still further derived from the living Leucocyte. The parasitic Symplast replacing the former nucleus stretches itself far out in band-like formations, so that there is a certain impression of an "albumin-melting into Fibrine"; in any case, in such a progressed dissolution there is, a priori, no explanation for such a picture unless one can bring to it the required patience for observing the process. The Chondrite-net system resulting from the host-cell, is especially developed toward the front, in the object at hand, and involves five Erythrocytes, with the marginal Symprotites of the parasites thereof the individual Symprotites copulate. In the main portion behind the "Pseudofibrine" as parasitic Symplast, the Symprotites have to the largest portion evolved probaenogenetically, and that is, into well-developed Thrombocytes with 3-7 Mych each, which to the largest part are far scattered and thus lost to the total picture.

The Lymphocyte pictured in Figure 6 shows that the cell-plasma-material of the host-cell can be nearly totally transmuted into Thrombocytes even in a Lymphocyte. In this picture, the nucleic material is changed into Symplast and pervaded with vacuola, the cellular plasma material of which, however, is almost completely without Chondrite-remnants. Only above the left center, a short splitting dumbbell -- consisting of two end-seeds (Symprotites) and a connecting filum -- projects from the nuclein Symplast; another one is likewise on the right center.

 
 
Figure 5. Blood of Cattle-Leucosis of 3/24/54 (Cattle #58a). Extensive Leucocyte destruction and dissolution. Host nucleus used up by the parasitic Fila (formerly considered to be "Fibrine"). Cell-plasma toward the front used up more by Chondrite-Dendroids; behind them, the Parasite-Symprotites have predominantly changed into Thrombocytes, respectively have evolved probaenogenetically (nucleus remains bluish; Thrombocytes pinkish in the applied Giemsa-coloring).   Figure 6. Lymphocyte remnant from Cattle-Leucosis 4/25/54 (Cattle #58a). Nucleic Symplast with small vacuola. Nucleic plasma of the host-cell completely transmuted into Thrombocytes of the parasite.

 

Figure 7. Lymphocyte remnant from blood with Cattle-Leucosis dated 4/25/54. Nucleic Symplast degenerating, without vacuola. Nucleic plasma completely transmuted into Thrombocytes by the parasite. Above, an Erythrocyte in the condition of expelling of two short rods of the Endobiont (Mucor racemosus Fresen) in the bacterial phase.

A very similar picture of far-reaching decomposition of a cell is shown in Figure 7 showing a Leucocyte-remainder in which the Thrombocytes are already about to break through the walls of the original host cell. The Erythrocyte located above is in a condition of expelling double-rods of the Leptotrichia bucchalis (Robin 1879), which are linked in chains of two's. With closer observation one recognizes that both are preparing for a splitting into two Thrombocytes each. The stretched wound of the Erythrocyte generally heals up and regenerates into a useful Erythrocyte. Even Erythrocytes that have 6-8 or even more such wounds on their edge, tend to complete themselves again and arrange themselves into the life-cycle of the host.

Figure 9. Fibrine-formation of Cattle-Leucosis in a direct blood-smear. The lower formation is the remnant of a Leucocyte-Dendroide, the upper a flocky Symplast composed of Chondrites and Protites. Cattle #37. Kiel 3/18/54.

If Arneth had removed a single such item, which he considered to be degenerated Thrombocyte-bacterial-forms, and cultivated it, he would have found that they are Leptotrichia buccalis, that is, the bacterial phase of the Mucor racemosus Fresen.

But, that the Endobiont of Cattle-Leucosis can reveal further possibilities for forming coarser "Fibrine-parts", by observation of direct blood-smears, is shown in Figure 9.

The lower portion is of a Leucocyte destroyed by an Endobiont. In the upper portion one recognizes a conglomeration of flocky "Fibrine" portions, seemingly representing Symplasts which originated through symplastisms of Protites and Chondrites. As is usually the case, one can notice projected Chondrite-threads which are also composed of Filum and Symprotites, and which always easily document the genetic nature of the materials from living albumin-masses of the Endobiont.

That the Cattle-Leucosis culminates finally in Sclero-Symplast formations, which completely correspond to the Felty-Syndrome in the human disease-form of the Endobiosis in this direction, is proven by the manifestations in Figures 10 - 13. They all stem from the cattle "Robert" from Kiel (blood-test #1) of 4/28/54, which was treated isopathically with Endobiont-Chondritine, which worked in spite of the conspicuously serious case. Figure 10 shows a hollow place between Erythrocytes, where Sclero-Symprotite-Symplasts began to form themselves, that there were remnants of the Leucocyte as Chondrite and Thrombocytes. In Figure 11 the Lymphocyte, completely destroyed by the parasite, was shrunk and the created hollow was crossed by Chondrite-threads; the plasma of the Lymphocyte was a lively blue in the Giemsa-coloring, and strongly light-refracting. Around this Leucocyte, Erythrocytes crowd around in the usual way and contain in the further surroundings 5 parties of red, increasingly deteriorating dendroid remnants of the Leucocyte-cores.

 

   
Figure 8. Further deteriorating Leucocyte-remnants, based on cattle-blood with Leucosis, 3/25/54. 3 nuclear remnants, nuclear Symplasts, the frontal one with Chondrite-Dendroid-Net. Cellular plasma material transmuted into larger Synascites, all of which are recognized as the forms of Leptotrichia buccalis (Robin 1879).   Figure 10. Cattle-Leucosis with beginning formations of Sclero-Symprotite-Symplasts.   Figure 11. Lymphocyte with strong light-refracting blue plasma. Adjoining hollow crossed by Chondrite-threads. 5 parties of red increasingly collapsing dendroid remnants of Leucocyte nuclei (a-e).

Illustrations 12 and 13 demonstrate the yet further unfolding processes of complete destruction of materials through the parasite.

 
Figure 12. Leucocyte with lively blue plasma (drawn in tiny points) and cubic-shaped Sclero-Symprotite-Symplast. The latter is colorless and strongly light-refracting. The three Thrombocytes above, with 4 or more Mych each: red. Nucleic remnants partly transmuting into an Erythrocyte.   Figure 13. A large chunk of Symplast, being dry albumin of the Endobiont. With nine blue enclosures of remnants of Leucocyte nuclei. A) loose red core of a Leucocyte, b) loose red core of a Lymphocyte.

The result of this sketch is that the Cattle-Leucosis is an intermediary form between cancer, Hodgkin, and Felty-Syndrome. The glandular swellings may grow to a very large extent, but are not as easily noted as in human beings. Even in very numerous blood-samples from sick and healthy cattle, the diagnosis of Leucosis has not been an easy one at all, when no blood-examinations are added. Occasionally, one finds even amongst healthy cattle herds some cattle with most serious cases of Leucosis -- but only through blood-diagnosis. The losses through Cattle-Leucosis are very high, namely 10%.

Figure 14. Blood from Leucosis-bull #25 (12/4/1954). a = frontal seam of a very very bright colloidal Symplast. Between the Erythrocytes are numerous sclerotic Chondrites. The regularly occurring lighter areas show in the dark-room larger colloid collections.

Sclero-Symplast-formations are also found in the blood of horses having the "Bornaic Disease", as has been described by me in an article, "Systatogeny, the Miracle of Rhythm of the Constructing Colloidal Happening at the Extreme Foundations of Life"; it is illustrated in Figure 53 [not shown]. As a post-script, Figure 14 shows the picture of the venous blood of the Leucosis-bull #25. It contains very large and very light colloid-symplasts, of which one is pictured below under a, the frontal seam of such a one. In the darkfield, one observes numerous colloid-collections in the bright larger areas of Leucosis blood, which happens not rarely, occurring island-like, which use up decomposing material of Erythrocytes for their own construction. Above left, several Sclerotic Systases, also in dry albumin form. These light areas, which occur regularly in the blood, containing colloid-collections, are an extremely typical picture in Leucosis, and they happen very frequently.

Causes Obstructing Insights in Technical Perspectives

  1. The saying: "Only an exact photo-copy can bring proof" is one of the most fundamental errors because such pictures present so many purely optically created, fake lines and elements which are never present in the pictorial recognition where there is constant physical micrometer adjustment.

    The experienced observer is simply used to seeing "plastically", "bodily." Not only the experienced microscope-reader, but any non-specialist, stands often enough helpless or even completely puzzled before such photographic pictures, for, recognition results from the observing, spatially plastic perspective based on years of experience.

  2. A very large portion of biologic growth processes involving comparative-morphologic elements lie underneath the cell and pertain primarily to the Symprotite, involving atomic-physical and quante-biological happenings, representing colloidal processes. Thus, in the growth of the Chondrite-stage we are dealing mostly with colloidal processes which happen all of a sudden in jumps of fractions of seconds -- thus causing the error-judgment that they are processes in albumin-coagulation and not growing processes.

 

Reasons Working Against an Understanding of Probaenogenetic Developmental Processes of Colloidal Living Elements of Primitive Phases as Albumin-Coagulation of a Purely Mechanical Nature (Fibrine).

All the following condemned erroneous concepts do not jibe with the biologic and veterinary disciplines or the fermentation micro-biology:

  1. An albumin-coagulation would comprise all blood elements equally! They would not start from single seriously-attacked morphologic elements and would not merely include the blood elements lying nearest.

  2. The "Fibrine-conception" existing for much longer than a century is a mere pseudo-science. These processes have never been doctrinally examined and compared with the natural growth-processes of the microbes beneath the cellular elements -- ,that is, their primitive phases, the elemental factors of which are composed of a homogenous
    unorganized and frequently immovable tiny albumin clump in colloidal forms. And whenever such comparisons were drawn upon, then the ruling disciplines repressed them as "Paroles".

  3. The fundamentals established by Bizzozero 1882 regarding the existence of blood-platelets (Thrombocytes) are, for the same reasons, also pseudo-science! Although later, these were taken to be metabolic products of Erythrocytes (Schilling, Wright, etc.), and Thrombo-kinasis was brought in as causal reason for it being a primal ferment promotive of blood clotting. But in reality, the Thrombocytes represent a special manifestation of the microbic growth-form "Thecite", and especially of the Chondrite-Thecite, which however, shows a great number of Mych -- which in the normal Thrombocyte are reduced to 3-7 Mych. Arneth (Qualitative Bloodlore, 1945) has rightly pointed out the too large and numerous Thrombocyte-forms which are aberrant or rather, in a pathogenic sense, count among the higher forms of Thecites, which not rarely show, for instance, even Synascites. If one extracts such Synascites and cultivates them, then one sees with absolute surety that they are purely cultures of Leptotrichia bucchalis, that is, a bacterial phase of Mucor racemosus Fresen, whereby the Synascites represent the bacterial forms in which the Mych are not lying one behind the other (catatact Ascites) but they come to lie in all directions of space in the usually much thicker and plumper bacterial rods. Therefore, if one takes the Thrombocytes to be metabolic products of Erythrocytes -- in reality they are the causal reason for the break-down: namely the parasite; on the other hand, it may be all kinds of cells which are, both in their cell plasma and in the nucleus, destroyed by parasites in Symprotite-form; and also these Symprotites subsequently mutate probaenogenetically into Thecites, which either already contain the Thrombocyte-form, or they swell up into larger Thecites, which subsequently fall apart into single Thrombocytes. These pictures are abundantly observable in blood which is stricken in this direction. But it can also be proven in many other places in the body, especially in the cells of the bone-marrow in which the giant cells and Megacaryocytes present tissue-cells which, through excessive attack of parasitic elements, first lose their capacity to split the cells and finally the entire ability to split the nucleus. Thus they grow into giant monstrosities which are stuffed full of parasites.

Additionally it is important for our own time -- in which one thinks globally about possibly supplying artificially cultivated fungi for human nutrition to supplement protein deficiency -- to point out that the meat of leucosis-diseased cattle must be legally forbidden as nutrition for humans; the everywhere prevailing idea of the Monomorphism of microbes has created catastrophic backwardness and incapacity for understanding. Because, in reality, it contains nothing other than forms of the Symbiont of all mammals, namely the Mucor racemosus Fresen, which is present in all meats in coarse measure.

A Few Aphorisms:

There is but one reliable Primal Science about nature and that is nature herself.
Insight becomes darkened when only small, ready-made sections limit the vision.

G.E. 1931

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