Potentiated Vaccine Treatment of Tuberculosis

© Copyright 2003 for the Estate of Professor Dr. Günther Enderlein, Germany
(Explore Issue: Volume 12, Number 3)

After VAUDREMER (1921), Tissot (1925) and other researchers had established the genetic relationship between the tubercle bacillus and molds of the genus (Aspergillus TBC–CHONDRITIN; formerly NIVELLANTIN), in 1930 I was able (with no knowledge of the work of the above-mentioned authors) to prove that the tubercle bacillus represents the bacterial development form of the Aspergillus mold, and that above all Aspergillus niger van Tieghem especially manifests itself.

FONTES (1910 and 1931) was the first to designate an invisible, filterable virus as the agent of tuberculosis. He and NICOLLE (1931) already supported a kind of Pleomorphism that, in addition to the normal bacillus, also postulated an invisible, filterable virus.

About a quarter-century ago, HOLLOS (Budapest) and PONCET (Paris) had assumed a special tuberculosis intoxication, and related this to the extended disease complex that we nowadays call paratuberculosis or lymphatic tuberculosis, etc.

I traced this intoxication back to the Chondrit stage (see Bacteria Cyclogeny for research).

In the Réunion (meeting – NR) of the 7th of November, 1948 in Paris, Mr. Léon JOLY reported in a comprehensive lecture on the history and significance of the remedy generally known in France as ART and in Germany as turtle vaccine [SVS. - Enderlein, (Mycobacterium phlei)1].

Although the internal effective mechanism of this remedy has not yet been fully clarified in all its details, I pointed out in 1931 that the entire defense apparatus is mobilized.

The successes reported at the November 7th, 1948 Réunion concerning the intracutaneous injections of ART vaccine carried out in France suggest that the skin is substantially involved in the defense process.

Here, we encounter the sizable topic area of cutivisceral reflexes in the HEAD sense. Proof of this can be derived from the fact that, in dermal tuberculids (efflorescences), the genuine acid-resistant tubercle bacillus is not to be found, but instead its primitive Chondrit stage – and, furthermore, the non-acid-resistant form in cases of lupus and tuberculous osteitis, and the nonbacterial primitive stages in acne cases. In these cases, the local defense processes potentiate themselves with the remotely-acting ones.

Besides this direct defense, the turtle bacillus Sclerothrix antituberculosis Enderlein 1931 has the ability to absorb the enzyme toxins of other bacteria.

This assertion stems from the observations of A. LESCHKE. Finally, the following should be noted: the ability of the turtle bacillus to influence the life of other – even pathogenic – bacteria. This is expressed e.g. in the fact that, like the real tubercle bacillus, it is able to react to the appearance of other bacteria by initiating unusually vigorous hypergenesis (multiplication) – thus driving out the other bacteria.

Summing up, I would like to outline the action mechanism of Sclerothrix antituberculosis as follows:

1. Direct stimulation of specific defense processes via the cutivisceral reflexes (diencephalons)
2. Ability to absorb the enzymatic toxins of other bacteria
3. Ability to drive out other bacteria through its own hypergenesis